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In vitro , 7-KC was observed to inhibit the expression of ABCD1 and homologs, including ABCD2 and ABCD3 in microglia and oligodendrocytes, resulting in peroxisome dysfunction, the root of disease occurrence

In addition, adenosylcobalamin is another metabolically active form of vitamin B 12 to be used as a cofactor for the enzyme methylmalonyl CoA mutase, which converts methylmalonylCoA to succinylCoA [14], Of note, a deficiency in vitamin B 12 leads to a functional loss of folate, which results from folate being trapped in the form of methyltetrahydrofolate [15]

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