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To investigate whether VRK2 directly phosphorylates Sec24C, we carried out an in vitro kinase assay and found that overexpression of VRK2 was able to increase the phosphorylation level of Sec24C

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In summary, our in vitro results support our hypothesis that LDHB, analogous to FSP1, transports reducing equivalents of NADH to CoQ into the lipid bilayer, thereby facilitating CoQH2-mediated inhibition of lipid peroxidation

A divergent astrocytic response to stress alters activity patterns via distinct mechanisms in male and female mice