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M2 tumor associated macrophages (TAMs), as a central regulator of the tumor microenvironment, affects tumor development, angiogenesis and immunosuppression

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We showed that glutathione (GSH) levels were reduced in erlotinib-resistant NSCLC cells in a T790M-dependent manner due to the decreased expression of GSH-synthesising enzymes

GPX4-IN-3 (26a) exhibits outstanding GPX4 inhibitory activity with a percent inhibition up to 71.7% at 1.0 M compared to 45.9% of RSL-3 [1]

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