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224,225 In Cu-deficient animals and humans, cardiovascular lesions could be presented as aortic fissures and aortic rupture, possibly due to an altered architecture of elastic fibers

Available research consists primarily of laboratory and animal studies, with limited controlled human data

Reversion of hypertrophic cardiomyopathy in a patient with deficiency of the mitochondrial copper binding protein Sco2: is there a potential effect of copper

291, R1120R1128

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There are also 2 autosomal recessive cytochrome b -positive forms of CGD identified as cytochrome b -positive CGD type I and type II