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33,34 The role of genetically edited mouse models in unraveling liver regeneration mechanisms Before the advent of genetically edited animal models, a conclusive demonstration of the functions and mechanisms of these growth factors was difficult, as researchers lacked the tools to specifically enhance or inhibit them in vivo

Am J Physiol Cell Physiol 322:C12-c23

5), and by inference, neurotoxic damage.53 Forgione and colleagues had previously hypothesized that GPX1 deficiency directly induces an increase in vascular oxidative stress, with attendant endothelial dysfunction.54 Glutathione peroxidases, particularly GPx1 have also been implicated in the development and prevention of many common and complex diseases, including cancer and cardiovascular disease (Fig

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402,403,404 Adipose tissue hypoxia and inflammation may lead to adipocyte dysfunction and obesity-induced insulin resistance in HFD-fed models, as indicated by increased infiltration of adipose tissue macrophages (ATMs), activation of the NLRP3 inflammasome and increased levels of proinflammatory cytokines

Amino Acids 42, 171179