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Raijmakers MT, Steegers EA, Peters WH

Metformin inhibits advanced glycation end products (AGEs)-induced renal tubular cell injury by suppressing reactive oxygen species generation via reducing receptor for AGEs (RAGE) expression

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Lack of coupling between the NADP redox status and E GSH in glr1 cells, provides a powerful genetic system to separate NADP and E GSH -dependent responses