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In this study, the authors show here that orally active PPAR activator GW-501516 significantly reduces fatty plaques in the arteries of animals

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MAPKK induces c-Jun N-terminal nucleated kinase (JNK) and p38 to translocate the nucleus inducing activator protein-1 (AP-1) transcription of pro-inflammatory molecules and processes (78, 79)

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Drug Content Uniformity Any standard assay method specified for the specific API in any of the standard Pharmacopoeia can be used to determine this

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