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and therefore it is a frequent contributor to cellular transformation, when normal function is compromised via genetic or epigenetic modifications

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By occupying GLP-1 receptors in the brain, the medication promotes insulin release, slows gastric emptying, reduces glucagon secretion from the liver, and affects the hunger control centre (lateral hypothalamus) of the brain to suppress appetite

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Early work characterized stability in human gastric juice and resistance to enzymatic degradation under acidic conditions (Sikiric et al., 2011)

10.1038/s42003-022-03877-w 67 LizarragaL