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Insofar as there is a contradiction between the effect of nicotine on regeneration and pain, it is essential to understand whether it is possible to block the development of pain without inhibiting nerve regeneration, in a condition in which such regeneration is desirable at an early stage of nerve injury

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This predicted Nuclear protein 1 (NUPR1) as a novel key regulator in these cells which prompted us to study the role of NUPR1 in phBECs including those derived from IPF patients