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This is due partly to cumulative effects of oxidative stress, which can damage DNA and impair oocyte viability

The past 10 years, however, have seen the development of enzyme therapy for arginase deficiency and gene therapy for ornithine transcarbamylase deficiency

Amyloid beta-peptide impairs ion-motive ATPase activities: evidence for a role in loss of neuronal Ca 2+ homeostasis and cell death

Despite evidence of systemic iron and ROS accumulation in FRDA, whether neuronal loss is directly mediated by ferroptosis remains an open question requiring further mechanistic exploration

NeuroImage 152, 1218