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With special reference to the novel mitochondrial disorder we have described in the CESLAC patients, several lines of research suggest that the ketogenic diet may be particularly useful for complex I defects (229, 230)

These findings suggest a microbiome-related approach may improve certain aspects of the condition but underline the complexity of addressing fatigue specifically [44, 78]

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Its important to maintain realistic expectations

196 A recent study found that the C/EBP/AEP signaling is age-dependently activated in human -synuclein transgenic mice and PD patients, which is responsible for mediating microglial activation and PD pathologies

however, mipomersen is unfortunately no longer available for clinical use, due to critical adverse effect of hepatotoxicity