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Increasing evidences document that necroinflammation is regarded as the essential pathological process in graft injury, and ferroptosis is pyramidally being proved to relate to transplantation-induced necroinflammatory response

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Of note, SLC7A11 deficiency has no effect on the anti-tumor activity of T cells, but inhibits mouse T cells proliferate in vitro ( Lipid Metabolism and Ferroptosis Interact to Regulate Tumor Immunity Microenvironment Lipid metabolism is tightly linked to the cell sensitivity to ferroptosis (Figure 2)