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Disclosures: Alina Jucov: Nothing to Disclose, Edward Gane: Nothing to Disclose, Man-Fung Yuen: Nothing to Disclose, Kosh Agarwal: Nothing to Disclose, Anca Streinu-Cercel: Nothing to Disclose, Young-Suk Lim: Nothing to Disclose, Keith Boundy: Nothing to Disclose, Cara Pilowa: Nothing to Disclose, Michael Chattergoon: Nothing to Disclose, Andre Arizpe: Vir Biotechnology: Employee, Vir Biotechnology: Stock - publicly traded company, Vaidehi Thanawala: Nothing to Disclose, Shuli Yu: Nothing to Disclose, Pan Wu: Nothing to Disclose, Yimeng Lu: Nothing to Disclose, Todd Correll: Nothing to Disclose, Tarik Asselah: Nothing to Disclose 417 THE IMPACT OF STEATOTIC LIVER DISEASE ON HBV INTEGRATION IN PATIENTS WITH CHRONIC HEPATITIS B VIRUS INFECTION Zhiwei Chen 1 Yan Han 1 Na Wu 1 Jing Tang 1 Peng Hu 1 Hong Ren 1 , 1 Second Affiliated Hospital of Chongqing Medical University Background: Steatotic liver disease (SLD) was associated with an increased risk of HCC among patients with chronic hepatitis B virus infection (CHB) but with a higher chance of achieving an HBsAg loss

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These results confirmed that the enzymatic reaction catalysed by Pt GSTF8 terminated the hydration equilibrium between flavan-3,3,4-triol ( 4 ) and flavan-3-on-4-ol ( 7 ), leading us to propose a GST-dependent dehydration of flavan-3,3,4-triol ( 4 ) or a tautomerization of flavan-3-on-4-ol ( 7 ) to form flav-2-en-3,4-diol ( 8 -B4), an equilibrium form of anthocyanins ( 1 ) in solution (Fig

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Os principais mecanismos descritos na literatura cientfica incluem: 1

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