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10.1002/chem.201404224 46 Sirgedait-ienV.StriganaviitG.ilanskienM.KniuipytI.PraspaliauskasM.VakeviienI.et al (2025)

at 20 g/mL, HRE showed the most pronounced effects by inhibiting proliferation, inducing G2/M cell cycle arrest, and promoting apoptosis

IB analysis of indicated proteins in HCT116 cells treated with or without SMART1

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This activation of the NRF2/SLC7A11/GPX4 pathway enhanced GSH levels, decreased lipid ROS accumulation, inhibited ferroptosis and lipid peroxidation, and improved the cognitive functions of AD mice ( In summary, Ghrelin, Neuritin, GAA, and Artemisinin may mitigate cerebral neuronal injury and cognitive dysfunction by modulating neuronal ferroptosis, lipid peroxidation, the antioxidant system, oxidative stress, mitochondrial dysfunction, and neuroinflammation

In AuDHD, both often operate simultaneously