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HDL, apoA-I, and endogenous apoE reduce lesion formation by preventing endothelial cell activation, inflammation, and oxidative stress and also by promoting cholesterol efflux from foam cells

Whether you are interested in the cellular repair mechanisms or the impact on mitochondrial health, the research continues to evolve

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This protective response involves up-regulations of the GSH-synthesizing enzyme, GSH-synthetase, the GSH-conjugating enzyme, GST, and the hepatocellular efflux pumps, Mrp2 and Mrp1

Madsen CM, Varbo A, Tybjaerg-Hansen A, Frikke-Schmidt R, Nordestgaard BG