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in addition, we found that clinical data support the role of transferrin in protecting against liver fibrosis by blocking ferroptosis, providing a possible therapeutic target for preventing ferroptosis-induced liver fibrosis [26]

This creates a dual surveillance system: GPX4 protects cytosolic and plasma membrane compartments, while DHODH-CoQH 2 specifically safeguards mitochondrial membrane integrity ( Beyond the core mechanisms described above, mounting evidence has revealed that multiple signaling pathways are involved in modulating ferroptosis