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(See PRECAUTIONS: Pediatric Use)

Studies in apoE-deficient mice expressing amyloid protein precursor (APP)-V717F demonstrated that apoE is actually required for amyloid plaque formation, at least in mice (Bales et al., 1999)

Nutrient solution was maintained full in the container, and a ventilation pump ensured the solution had enough oxygen

This mechanism proved to prevent the overexpression of mitochondrial structural genes, thus preserving mitochondrial functions and protecting cells against A toxicity (Manczak et al