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Demonstrated that BPC-157 improves insulin signaling and reduces TNF- and IL-6 release in hepatocyte cultures exposed to high-glucose conditions, providing a new model for studying metabolic-dysfunction pathways

Leveraging the presence of a disulfide bond, we posited that cross-linked structure could impede the premature seepage of CUR and miR155 in a physiological environment

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The BDNF Val66Met polymorphism moderates the relationship between posttraumatic stress disorder and fear extinction learning

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