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Immediately following mitotic exit, APC/C shifts to using a second substrate adaptor, the Cdc20-related protein Cdh1/Fzr1 (hereafter referred to as Cdh1)

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Toxicol Sci 180:2637

By removing the damaged mitochondria, mitophagy prevents further oxidative stress and the potential onset of lipoapoptosis, a form of cell death triggered by excessive lipid accumulation [86,87,88],

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