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The angiogenic switch can be triggered either by additional genetic alterations of tumor cells, leading to increased proliferation and hypoxia or expression of pro-angiogenic factors, or by tumor-associated inflammation and recruitment of immune cells

MS raw files and the corresponding results files, including databases, were deposited to the ProteomeXchange Consortium via the PRIDE partner repository with the dataset identifier PXD069482

To investigate the mechanism underlying the effect of GSH on TGF signalling, we next detected TGF1 protein expression and SMAD3 phosphorylation

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Cystinosis stands as a prototypic disorder for a subgroup of lysosomal storage diseases caused by defective lysosomal transporters [2]