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The above evidence indicates that AGNHW alleviates cerebral edema and exerts neuroprotective effects by synergistically regulating vascular permeability, BBB integrity, and inflammatory responses, providing a multidimensional pharmacological basis for the prevention and treatment of HACE

Dysregulated neurotransmission further impairs the brains ability to form and retain memories, highlighting the crucial link between biochemical alterations and cognitive symptoms (Handra et al., 2019)

The highest concentrations of APP at steady state are found primarily at the TGN and early endosomes (Caporaso et al, 1994

Saiga AI, Iwai K, Hayakawa T, Takahata Y, Kitamura S, Nishimura T, et al