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Intention of the article is the collection of data to support the view that Wilson disease is a complex multifaceted and unique copper overload condition, where intracellular accumulation of toxic free copper, but not total copper, causes organ damage

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Furthermore, immune dysfunction leads to altered balance within the immune pathways resulting in excess inflammation

Correlations between gene modules and experimental conditions (genotype or treatment) were tested using the limma R package, with multiple testing corrections via the BenjaminiHochberg method