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Neuroinflammation in ME/CFS was initially suggested by studies showing elevated levels of pro-inflammatory cytokines, and decreased anti-inflammatory cytokines, in cerebrospinal fluid from lumbar punctures in ME/CFS patients [62, 63], and further supported by a positron emission tomography (PET) study [64], which showed increased binding of a radioligand to 18 kDa translocator protein (TSPO) receptor, the expression of which increases when microglia are activated

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It is used for reconstituting lyophilized peptides and maintaining sterility for multi-use vials

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These molecules trigger ferroptosis by different methods, GSH depletion, GPX4 inactivation, or degradation (13, 29)