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Preliminary evidence on safety and clinical efficacy of luteolin for patients with prostate cancer under active surveillance

Although we agree with the authors of previous studies who have suggested that reduced synthesis of GSH induced by liver dysfunction and increased oxidative stress might cause HCC tissue to lose its capacity to provide protection against oxidative impairment [1,11,25,31], this cannot explain why our HCC patients had lower MDA and higher GSH levels in HCC tissue when compared to its adjacent normal liver tissue

Mechanism for manganese enhancement of dopamine-induced oxidative DNA damage and neuronal cell death

Vogler M, Braun Y, Smith VM, Westhoff M-A, Pereira RS, Pieper NM, et al

[25] [26] Anti-thyroid drugs [edit] Treatment with methimazole and propylthiouracil for hyperthyroidism, is commonly associated with an elevation of liver enzymes

This pathway is a major way the body maintains glutathione levels and manages sulfur metabolism