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Description

Silencing of FABP1 ameliorates hepatic steatosis, inflammation, and oxidative stress in mice with nonalcoholic fatty liver disease

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When DNA damage is present, p53 is crucial in triggering cell-cycle arrest or promoting programmed cell death (apoptosis) [22]

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Advances in microbiome research may allow personalized interventions targeting dysbiosis patterns associated with specific diabetic complications

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