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Targeting redox homeostasis in rhabdomyosarcoma cells: GSH-depleting agents enhance auranofin-induced cell death

Having elucidated the ferroptosis-immune-metabolism axis as a key self-sustaining mechanism in COPD pathogenesis, these insights provide a foundation for novel therapeutic interventions

Researchers have focused on regulation of fatty acid anabolism in T cells [45, 46], and interestingly, different studies have reported opposite results

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Oxidative stress can broadly interfere with enzymes involved in glycolysis, the TCA cycle, and ATP biosynthesis, ultimately leading to glucose metabolism disorders and reduced ATP production [70, 76]