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Its probable mechanism includes activation of renal AGE-RAGE signaling pathways, decreasing levels of monocyte chemotactic protein-1 (MCP-1) and PKC-A, which consequently reduces the production of slit diaphragm proteins such as nephrin and podocin, thereby mitigating the impact of AGE-mediated DN pathogenesis (Muthenna et al., 2014)

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308 , F1259F1267 (2015)

doi: 10.1002/eji.202350901 20 CeppelliniRGarottaGMalavasiFTruccoM

( PspXI * ) and Lachnoclostridium phytofermentans ( LpXI * ), coupled with native xylulokinase ( XYL3 ), were introduced