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Indeed, a transgenic syn/amyloid/tau pathology mouse line (DLB-AD), established by crossing the 3xTg-AD [161] amyloid/tau line with the M83-h [216] mutant A53T syn line, demonstrated that syn pathology is capable of promoting A and tau accumulation [3]

By comparison, the neuroprotection appeared in a mutant alpha-synuclein (Syn

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(A) trr1 and tpx1 thioredoxin mutant strains were sensitive to H2O2 compared to WT on CM at the concentrations shown

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