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Modifying gene expression across antioxidant, apoptotic, metabolic, and genomic stress pathways reinforces its promise as a natural therapeutic agent

Acknowledgements The authors are grateful to the International Centre for Genetic Engineering (CRP/ NGA18-02, ICGEB) Italy, for the grant awarded to Amos Abolaji for the purchase of equipment, materials and resources used to carry out this project over the past years

We found that H19 mainly regulates oxidative stress and cell-cycle genes and the primary route of cisplatin resistance involved oxidative-stress pathways, especially NRF2-targeted genes in the GSH pathway (Fig

Sugere-se que a manuteno dos nveis normais de carnitina podem inibir a perda de massa muscular, a lipognese heptica, a hipertrigliceridemia e a diminuio da oxidao de cidos graxos

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