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The animals were allowed a diet and water ad libitum

Clinical Information a familial form of congenital hyperbilirubinemia transmitted as an autosomal recessive trait

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Mutations in GBA1 represent one of the most common genetic risk factors for PD, leading to significantly reduced GCase protein activity in cholinergic neurons and impairing -syn metabolism, thereby elevating -synuclein levels in these neurons [179]