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Once released, these factors can interact with intracellular antiapoptotic and survival-promoting proteins, inducing mitochondria-mediated apoptosis

ATP as a pathophysiologic mediator of bacteria-host crosstalk in the gastrointestinal tract

Bishehsari F, Magno E, Swanson G, Desai V, Voigt RM, Forsyth CB, Keshavarzian A

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This pattern was observed for 109 metabolites, of which 44 did not differ between IR phenotypes when comparing iAUCs between IR phenotypes, and were thus only identified as different when comparing curve shapes