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The progressive degeneration of dopaminergic neurons in the substantia nigra pars compacta leads directly to dopamine deficiency, underpinning the hallmark motor symptoms such as bradykinesia, resting tremor, and rigidity, as well as contributing to nonmotor symptoms including autonomic, mood, and cognitive disorders [2]

The presence of malfolded proteins in the endoplasmic reticulum signals the induction of glucose-regulated proteins

Therefore, improving neuronal function through TCA cycle interventions requires careful consideration of the impact of TCA cycle to prevent disruptions in intracellular signal transmission

This approach eliminates M.tb through mechanisms that bypass the pathways commonly targeted by conventional anti-TB drugs, thereby reducing the risk of developing antibiotic resistance [5, 7]

Single-Cell and Spatial Innovations in Peritoneal Carcinomatosis (Keynote Lecture)

FOXN4 affects myocardial ischemia-reperfusion injury through HIF-1/MMP2-mediated ferroptosis of cardiomyocytes