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Using this approach, several potential targets have been identified, including PRKACA, NCOA2, and PPARA, which play crucial roles in regulating neuroendocrine function, metabolism, and neuro-immunity [246]

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As an effector molecule, NO participates in antimicrobial responses and cooperates with oxidative stress processes to amplify inflammatory signaling, thereby being associated with an M1-like pro-inflammatory program ( + macrophage-like state with immunoregulatory features in mouse macrophage models ( In addition to generating terminal effector molecules, Arg metabolism can also influence inflammatory transcriptional programs through metabolic-signaling coupling