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However, the mechanism by which NQO1 mediates these functions is not yet well understood

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Acknowledgements The authors would like to thank Islam Elgohary, researcher in Pathology Department, Animal Health Research Institute (AHRI), Dokki, Giza, Egypt, for his efforts in revision of histopathological results

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146 SoderbergC.GillamM

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Kadota K, Takamura N, Aoyagi K, Yamasaki H, Usa T, Nakazato M, Maeda T, Wada M, Nakashima K, Abe K, Takeshima F, Ozono Y

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IP-10, the human homolog of MOB1, is overexpressed in most CRCs (Zhang et al., 1997)

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Its primary mechanism runs through AMPK activation

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