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In the absence of intracel-lular nucleophiles such as GSH, this reactive metabolite ( N -acetylbenzoiminoquinone) reacts with nucleophilic groups of cellular proteins, resulting in hepatotoxicity.The chemical and toxicologic characterization of the electro-philic nature of the reactive acetaminophen metabolite has led to the development of effective antidotescysteamine and N -acetylcysteine

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To test this hypothesis, we also measured glutathione redox potential and H 2 O 2 dynamics using Grx1-roGFP2 and roGFP2-Orp1, respectively

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Comorbid with inflammatory-mediated immune diseases (adjusted OR: 1.58, p = 0.066), particularly ankylosing spondylitis (adjusted OR: 3.38, p = 0.001), also increased stroke risk

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