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Intranasal drug delivery: opportunities and toxicologic challenges during drug development

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Receptor number and caveolar co-localization determine receptor coupling efficiency to adenylyl cyclase

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VCN managed to halt the progression of fibrosis either directly by interfering with the classical TGF-1/Smad 2/3 signaling pathway or indirectly by upregulating the expression of Nrf2, enhancing the antioxidant system, activating PPAR- and downregulating the NLRP3/NF-B pathway which influences the production of inflammatory cytokines involved in the pathogenesis of pulmonary fibrosis