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While normal cells preserve membrane localization of EAATs, their mislocalization in malignant cells together with up-regulation of xCT lead to aberrant handling of glutamate that induces high levels of glutamate in the extracellular space, as seen in gliomas (192) and in breast cancer, where endocrine therapy-resistant cells promote autophagy and glutamate import via EAAT2 (60, 193)

173 High expression of p65, SHh, and Gli1 was observed to be associated with poorer prognosis in patients with advanced prostate cancer

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Loss of hepatocyte growth factor/c-Met signaling pathway accelerates early stages of N-nitrosodiethylamine induced hepatocarcinogenesis

Efficacy and safety of Zicuiyin decoction on diabetic kidney disease: a multicenter, randomized controlled trial