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These alterations promote the production of nephrotoxic galactose-deficient IgA1 and exacerbate disease progression [139, 140]

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Median age at onset in patients with BRIC-1 was 12 years while BRIC-2 was 1 month

Pathogen triggered TLRs are generally understood to cause an inflammatory immune response, supporting the subsequent clearance of pathogenic microorganisms (Mayne et al., 2020)

Taken together, these results suggest regulation of GS may also be important in determining the overall GSH synthetic capacity under certain conditions and especially in non-hepatic tissues

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