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10.1002/j.1460-2075.1986.tb04528.x EMBO J

David Beck, MD, Ochsner Medical Center, New Orleans, LA

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Taken together, GH excess associated with poorly controlled T1DM induces podocyte injury, characterized by podocyte hypertrophy, apoptosis, dedifferentiation of podocytes (epithelialmesenchymal transition) and/or cross-linking of the basement membrane resulting in increased podocyte permeability to albumin and detachment of podocytes from the glomerular basement membrane (Fig