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Oral treatment with Cu(II)(atsm) increases mutant SOD1 in vivo but protects motor neurons and improves the phenotype of a transgenic mouse model of amyotrophic lateral sclerosis

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T cells activated by iDCs decreased the expression of heat shock proteins in tumor cells, increasing their heat sensitivity

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Abstract Inhibition of the protein kinase mechanistic target of rapamycin (mTOR) with the Food and Drug Administration (FDA)-approved therapeutic rapamycin promotes health and longevity in diverse model organisms

Therefore, while BV may have potential as a therapeutic compound, its safety in clinical trials is still a matter of concern