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doi:10.1093/tropej/fmi074

Oxidative stress can induce mitochondrial dysfunction in macrophages, facilitate conversion of macrophages to foam cells, and induce a pathological switching in the VSMC phenotype

By the same reasoning, interventions that directly inhibit Rlip could reduce inflammation, but promote apoptosis

doi: 10.1007/s11101-014-9336-6

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These compounds have demonstrated promising anti-cancer properties, including tumor immunity enhancement, reversal of multidrug resistance, regulation of autophagy and ferroptosis, and antiproliferative, pro-apoptotic, and anti-metastatic effects, both in vitro and in vivo (Fig