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It accelerates tendon fibroblast outgrowth, increases cell survival under stress, and promotes cell migration, likely through the activation of the FAK-paxillin pathway

This encompasses collagen induction, promotion of wound healing, antioxidant effects, inhibition of inflammatory cytokines, and promotion of skin repair

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5a), and the percentage of keloid fibroblasts in G0/G1 phase (90.78% 1.62%) was significantly higher compared with that in normal skin fibroblasts (75.47% 4.62%) ( p = 0.03