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The evidence base is preclinical for IGF-1 LR3 specifically, with adjacent human data from native rhIGF-1 (mecasermin/Increlex)

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Some models suggest reductions in visceral fat markers when Tesamorelin is examined in isolation, while additional studies propose that peptide combinations may contribute to modulating lipid and glucose-related pathways

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For example, the decreased expression of genes associated with aerobic metabolism and glycolysis in HCC cells can inhibit liver cancer growth [2]

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