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2007 [cited 2025 Apr 19];21(3):11330

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10.1016/j.ejcb.2015.05.002 [DOI] [PubMed] [Google Scholar] Lin W, Paterson GA, Zhu Q, Wang Y, Kopylova E, Li Y, Knight R, Bazylinski DA, Zhu R, Kirschvink JL, & Pan Y (2017)

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Mechanistically, DM-induced perturbations in the AMPK/mTOR signaling axis, impaired branched-chain amino acid catabolism, and tryptophan-kynurenine axis dysregulation collectively drive oxidative stress and immune homeostasis disruption through cascade reactions, culminating in pulmonary parenchymal injury

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Circadian disturbances are not likely to be an epiphenomenon of bipolar illness given that they are present during mania, depression, in euthymic state, and in healthy relatives of bipolar patients (181, 182)

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For example, in the Ec57 I site or CTGAAG(16/14), there is a portion of relatively high specificity (i.e

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Adding glucagon or GIP receptor activation adds mechanisms that enhance energy expenditure, improve substrate utilization, and modify fat cell function

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