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This suggests that the pathological processes and tissue damage established over years of CFTR dysfunction may not be entirely reversible by the current levels of CFTR correction achieved, or that the restoration of CFTR function is insufficient to fully extinguish these self-perpetuating cycles of inflammation and oxidative damage

The rescue experiment suggests that GSTM2 may be a useful therapeutic target for hepatic steatosis

doi: 10.1016/j.acthis.2023.152000

(2025) used plasma technology to generate novel superhydride donors that specifically upregulated SQOR in cardiomyocytes

doi: 10.3390/microorganisms8122018 51 KolodziejczykA

doi: 10.1016/j.ctcp.2016.11.008 149 ChenJGuoQY-HPRenQ-LChiLHuR-Ket al