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However, existing in vitro BBB models cannot mimic these dynamic pathological changes and spatial heterogeneity, leading to fundamental limitations in predicting drug permeability and the therapeutic window

fuscopurpurea cDNA library was 100%

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Interestingly, whilst the FFE689-91 residues appeared to be essential for SNX17 binding, the FFE689-91AAA mutant bound RABGAP1 with approximately a 3-fold increase

m/z , 3001800) were recorded in the Orbitrap, followed by higher energy collision dissociation (26% normalised collision energy) of the 10 most intense MS peaks

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