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Therefore, it is probable that TG2-mediated modification of GSTP1 affects the interactome of cellular GSTP1, resulting in an alteration of interactions with its binding partners

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The observed metabolic flexibility, particularly delayed glucose oxidation and compensatory substrate use, can be harnessed to model pathological states such as GLUT1 deficiency, neonatal hypoglycemia, or ketogenic interventions

doi: 10.1016/j.mib.2022.102237 194 KavousiK.BagheriM.BehrouziS.VafadarS.AtanakiF