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Ethics approval for animal experimentation was obtained from Washington State University

Disrupting the p53-FOXO4 interaction using FOXO4-DRI caused p53 to be excluded from nucleus and directed to mitochondria for induction of apoptosis, ultimately eliminating the senescent cells

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The precision of this mechanism is a key advantage, as it minimises collateral damage to surrounding tissues. (Peptide Gurus) Summary of findings In summary: FOXO4-DRI has been shown to reduce senescent cell number in animal models and cell culture

Open question Whether the same mechanisms are clinically meaningful in humans is not yet established