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There's no strong theoretical reason to weight angiogenesis more heavily than cell migration (or vice versa) in tissue-repair research both are prerequisites for repair, both contribute distinct mechanisms, and neither obviously dominates the other in published research models

Expanding the models application to various hospitals that use similar coding systems and incorporating broader datasets from different specialties would provide a more robust assessment of the effectiveness of embedding-based representations in diverse clinical scenarios

Another effect of IGF-I was insulin-like and not inhibited by insulin specific antibodies (2,3,4) and therefore it was named non-suppressible Insulin-like activity (NSILA)

The clearest indication is a diagnosed deficiency, because direct delivery corrects low levels faster and more reliably than oral replacement