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Description

This study reports a novel mechanism of NRF2 in repressing the expression of pro-inflammatory genes

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2 and Table 2)

Overall, we expect our Drug-ID/isASO-ID protocols to work for other classes of ON drugs, too 1,8 , including splice-switching ASOs, siRNAs, antimiRs and ADAR-recruiting ASOs 51

On the inspection of transcriptional level, it was unexpectedly to find that IGF-1 and EE significantly down-regulated MRF4 expression, since that down-regulation of MRF4 was reported in IGF-1 knockout mice 55